GSTT1/4 Polyclonal Antibody, 50ul Peripheral Blood Separation Tube Alternative splicing or deletion caused

Alternative splicing or deletion caused by a translocation event in this paternally-expressed region is responsible for Angelman syndrome or Prader-Willi syndrome due to parental imprint switch failure

plasmids often carry genes such as antibiotic resistance genes and fluorescent protein genes

The gene maps to the region on chromosome 22q11

The gene clusters demonstrate an unusual genomic organization similar to that of B-cell and T-cell receptor gene clusters

GSTT1/4 Polyclonal Antibody, 50ul Peripheral Blood Separation Tube Alternative splicing or deletion causedGlutathione S transferase theta 1 encoded by GSTT1 is a member of a superfamily of proteins that catalyze the conjugation of reduced glutathione to a variety of electrophilic and hydrophobic compounds. Human GSTs can be divided into five main classes: alpha, mu, pi, theta, and zeta. The theta class includes GSTT1, GSTT2, and GSTT2B. GSTT1 and GSTT2 GSTT2B share 55% amino acid sequence identity and may play a role in human carcinogenesis. The GSTT1